Researchers identify potential MASH target after gene disruption triggers liver symptoms in mice
Researchers at Waseda University in Japan have identified a gene that, when rendered nonfunctional in mice, causes the rodents to develop telltale signs of the liver disease metabolic dysfunction-associated steatohepatitis (MASH). The result suggests that the protein made by the gene, known as Nwd1, and other molecules it interacts with could be worthwhile targets for new MASH therapies.
